NSAIDs can cause _____ necrosis (kidney) as an adverse effect
Most NSAIDs increase risk of GI bleeding due to inhibition of _____
Dyslipidemia drugs _____ are the most effective lipid lowering medications for preventing future cardiovascular events
PGI2 _____ platelet aggregation
What drug intoxication presents as pupillary dilation, grandiosity, hypertension, and paranoia that lasts for multiple hours? _____
Neuro/Psych_____ is a -opioid inverse agonist used to treat acute opioid toxicity (can precipitate withdrawal)
Oral _____ contain high doses of estrogen and progesterone (or progesterone alone) which prevent ovulation via inhibition of FSH and LH
Alpha-glucosidase inhibitors (e.g. acarbose, miglitol) reduce the activity of alpha-glucosidases on the _____, thus delaying carbohydrate hydrolysis and absorption
Sudden discontinuation of SSRIs or SNRIs can cause _____-like symptoms
Barbiturates can cause severe CNS depression (e.g. coma) and should be avoided in _____ patients
Antiarrhythmic classification and mechanisms
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Class I antiarrhythmics (sodium channel blockers)
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Class II antiarrhythmics (beta blockers)
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Class III antiarrhythmics (potassium channel blockers)
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Class IV antiarrhythmics (calcium channel blockers)
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Other antiarrhythmic agents
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Antianginal drugs
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Positive inotropic agents
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Phosphodiesterase inhibitors
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Cardiac glycosides
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Vasopressors and inotropes
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Pulmonary hypertension therapies
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Peripheral vascular disease therapies
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