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Pharmacology
⭐ High Yield

Beta-blockers: Mechanisms and Clinical Use

8 min read42% done

Beta-blockers competitively inhibit catecholamines at β-adrenergic receptors. Selective β1 blockers (metoprolol, atenolol) primarily affect cardiac tissue, reducing heart rate and contractility.

β1 selective = cardiacContraindicated: asthmaLabetalol: non-selective
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Pharmacology›Autonomic Drugs›Beta-blockers
## Mechanism of Action
Beta-blockers competitively inhibit catecholamines at β-adrenergic receptors, reducing heart rate and myocardial contractility.
⭐HIGH YIELD
β1 selective blockers: metoprolol, atenolol, bisoprolol

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PHARMACOLOGY — Beta-blockers
## Receptor Selectivity
Cardioselective (β1) blockers have higher affinity for cardiac β1 receptors compared to pulmonary β2 receptors, making them safer in patients with reactive airway disease.
This selectivity is dose-dependent — at high doses, even cardioselective agents may block β2 receptors.
RezzyAsk Rezzyabout pulmonary β2 receptors
Why are β2 receptors important in asthma?
RezzyRezzy
β2 receptors in bronchial smooth muscle cause bronchodilation when stimulated. In asthma, blocking them (β2 blockade) causes bronchoconstriction → can trigger an attack. That's why cardioselective β1 blockers are preferred.
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