NEET-PG 2015 Practice Questions

Q: Which anatomical structure lies between the bulla ethmoidalis and the uncinate process of the ethmoid?

Ethmoidal infundibulum. ***Ethmoidal infundibulum*** - The **ethmoidal infundibulum** is a curved, three-dimensional space located within the **lateral wall of the nasal cavity**. - It forms a critical drainage pathway, often leading to the opening of the **maxillary sinus** and serving as the primary outflow tract for the anterior ethmoidal air cells. *Bulla ethmoidalis* - The **bulla ethmoidalis** is the largest and most constant anterior ethmoid air cell, bulging into the middle meatus. - It lies *superior and posterior* to the ethmoidal infundibulum, forming one of its boundaries, not the space itself. *Uncinate process of ethmoid* - The **uncinate process** is a sickle-shaped bony lamella that forms the *anterior and inferior boundary* of the ethmoidal infundibulum. - It helps define the pathway for drainage but is a bony structure, not the intervening space. *Maxillary sinus* - The **maxillary sinus** is a paranasal sinus located within the maxilla, which drains into the posterior part of the ethmoidal infundibulum or hiatus semilunaris. - It is a separate air-filled cavity, not the anatomical space between the bulla ethmoidalis and uncinate process.

Q: What lies between the middle and inferior turbinate?

Middle meatus. ***Middle meatus*** - The **nasal meatuses** are passages in the nasal cavity that lie inferolateral to a corresponding **turbinate**. - Therefore, the **middle meatus** is located directly beneath the **middle turbinate** and superior to the **inferior turbinate**. *Superior meatus* - The **superior meatus** lies below the **superior turbinate**. - It drains the posterior ethmoid air cells and the sphenoid sinus. *Hiatus semilunaris* - The **hiatus semilunaris** is a curved opening located within the **middle meatus**. - It is an important drainage pathway for the frontal sinus, maxillary sinus, and anterior ethmoid air cells. *Inferior meatus* - The **inferior meatus** lies below the **inferior turbinate**. - It receives the opening of the **nasolacrimal duct**.

Q: What is the narrowest part of the nasal cavity?

Internal nasal valve area. ***Internal nasal valve area*** - The **internal nasal valve** (ostium internum) is the **narrowest part of the nasal cavity**, located approximately 1.3 cm from the nostril - Formed by the **septal cartilage medially**, **upper lateral cartilage laterally**, **nasal floor inferiorly**, and **anterior head of inferior turbinate posteriorly** - The angle between the septum and upper lateral cartilage is typically **10-15 degrees**, creating the narrowest cross-sectional area - Accounts for approximately **50% of total nasal airway resistance** and is clinically the most critical site for airflow regulation - **Clinical significance**: Site of nasal valve collapse in breathing disorders *Vestibule* - The **nasal vestibule** is the most anterior part of the nasal cavity lined with **keratinized stratified squamous epithelium** and **vibrissae** (nasal hairs) - While it is a narrow region, it is **NOT the narrowest part** of the nasal cavity - Acts as the entrance to the nasal cavity but has a larger cross-sectional area than the internal nasal valve *Choanae* - The **choanae** are the **posterior openings** of the nasal cavity that open into the nasopharynx - They represent a transition point for airflow but are relatively **wide openings**, not the narrowest part *Inferior turbinate* - The **inferior turbinate** is a bony projection covered with erectile tissue that increases surface area for warming and humidifying air - While it can become engorged and narrow the airway pathologically, anatomically it does not constitute the narrowest fixed point of the nasal passage

Q: Which perforation of the tympanic membrane is most commonly seen with tubotympanic CSOM?

Central. ***Central*** - A **central perforation** of the tympanic membrane is the most common type seen in **tubotympanic chronic suppurative otitis media (CSOM)**. - This type of perforation involves the **pars tensa** of the tympanic membrane, leaving an intact annulus. *Anterosuperior* - While perforations can occur anywhere, an **anterosuperior perforation** is not the hallmark of tubotympanic CSOM. - This location does not specifically correlate with the characteristic inflammatory patterns seen in tubotympanic disease. *Posterosuperior* - A **posterosuperior perforation** is more often associated with **atticoantral CSOM** due to **cholesteatoma formation**. - **Cholesteatoma** typically begins in the pars flaccida or posterosuperior pars tensa, which is different from tubotympanic CSOM. *Posteroinferior* - A **posteroinferior perforation** is not the most typical presentation for tubotympanic CSOM. - This location does not specifically differentiate it from other forms of otitis media or reflect the primary pathology of tubotympanic disease.

Q: Most common cause of otitis externa is

Bacterial infection. ***Bacterial infection*** - The most common pathogens causing **otitis externa** are bacteria, primarily **Pseudomonas aeruginosa** and **Staphylococcus aureus**. - This condition, often called "swimmer's ear," is favored by moisture in the ear canal, which creates a conducive environment for bacterial growth. *Fungal infection* - While fungal infections (otomycosis) can cause otitis externa, they are **less common** than bacterial causes. - Fungi like *Aspergillus* and *Candida* are typically involved, often in immunocompromised individuals or after prolonged antibiotic use. *Seborrheic disease* - **Seborrheic dermatitis** can affect the ear canal and surrounding skin, leading to flaking, itching, and inflammation. - However, it is a primary skin condition and does not directly cause infectious otitis externa, although it can predispose to secondary infections. *Herpes Zoster* - **Herpes zoster oticus** (Ramsay Hunt syndrome) is a viral infection affecting the facial nerve, causing a painful rash, facial paralysis, and hearing loss. - It is a specific viral etiology with distinct neurological symptoms and is not a common cause of general otitis externa.

Q: Which of the following is a cause of objective tinnitus?

Carotid artery aneurysm. ***Carotid artery aneurysm*** - An **aneurysm** of the **carotid artery** can cause pulsatile tinnitus that is audible to others (objective) due to turbulent blood flow. - The sounds originate from vascular structures within or near the ear, making them detectable by a clinician. *Impacted Wax* - **Impacted cerumen** is a common cause of **subjective tinnitus**, where the sound is heard only by the patient. - It can muffle external sounds and alter the perception of internal body sounds, but it does not produce a sound audible to others. *Meniere's disease* - **Meniere's disease** is characterized by **subjective tinnitus**, along with vertigo, hearing loss, and aural fullness. - The tinnitus in Meniere's is typically a low-pitched roaring or buzzing sound, audible only to the affected individual. *Ototoxic drugs* - **Ototoxic drugs** (e.g., aspirin, aminoglycosides) commonly induce **subjective tinnitus** as a side effect. - These medications primarily damage inner ear structures, leading to perceived sounds that are not externally measurable.

Q: Hitselberger's sign is seen in?

Acoustic neuroma. ***Acoustic neuroma*** - Hitselberger's sign is an area of **hypoesthesia** or **anesthesia** in the **posterosuperior portion of the external auditory canal**. - Acoustic neuromas (vestibular schwannomas) arise from the **vestibular portion of CN VIII** in the internal auditory canal. - This sign results from involvement of the **sensory fibers of the nervus intermedius** (sensory branch associated with the facial nerve) due to close anatomical proximity in the cerebellopontine angle. - This symptom, also known as the **"ear canal sign,"** is relatively specific for acoustic neuroma. *Glomus Tumour* - Glomus tumors are **highly vascular benign tumors** of the **middle ear** that present with **pulsatile tinnitus** and conductive hearing loss. - While they can affect cranial nerves, Hitselberger's sign is not a characteristic presentation. *Nasal angiofibroma* - Nasal angiofibromas are **benign, highly vascular tumors** found in the **nasopharynx**, primarily affecting adolescent males. - They typically present with **epistaxis** and **nasal obstruction** and do not cause Hitselberger's sign. *Acute suppurative otitis media* - Acute suppurative otitis media is an **infection of the middle ear** causing earache, fever, and hearing loss. - It does not involve the cerebellopontine angle or sensory innervation of the external auditory canal in a way that would lead to Hitselberger's sign.

Q: Which of the following statements about vasomotor rhinitis is NOT true?

It is caused by an allergic reaction. ***It is caused by an allergic reaction.*** - Vasomotor rhinitis, also known as nonallergic rhinitis, is characterized by symptoms similar to allergic rhinitis but is **not mediated by an allergic reaction** or an IgE-mast cell response. - Its etiology is related to the **dysregulation of the autonomic nervous system**, specifically an imbalance in the vascular tone of nasal blood vessels, rather than an allergic trigger. *It is due to parasympathetic overactivity* - **Parasympathetic overactivity** is a recognized underlying mechanism in vasomotor rhinitis, leading to increased vascular permeability and glandular secretion. - This overactivity results in symptoms such as **nasal congestion**, rhinorrhea, and sneezing, mimicking allergic rhinitis without an identifiable allergen. *Resistant cases may need cryotherapy* - For severe and **medically refractory cases** of vasomotor rhinitis, **cryotherapy** (specifically cryoablation of the posterior nasal nerve) can be a treatment option. - This procedure targets the nerves responsible for parasympathetic outflow to the nasal mucosa, thereby reducing symptoms like rhinorrhea and congestion. *It may lead to hypertrophic rhinitis* - Chronic inflammation and vascular engorgement associated with long-standing vasomotor rhinitis can lead to **mucosal hypertrophy**, particularly of the inferior turbinates. - This condition, known as **hypertrophic rhinitis**, can exacerbate nasal obstruction and may require surgical intervention to improve airflow.

Q: Which of the following statements about vasomotor rhinitis is false?

It is an infective condition. ***It is an infective condition*** - **Vasomotor rhinitis** is a **non-allergic, non-infectious** condition of the nasal passages. - Its pathophysiology involves **autonomic nervous system dysfunction** affecting nasal blood vessels, not microbial infection. *It primarily presents with nasal congestion and rhinorrhea* - This statement is **true** because classic symptoms of vasomotor rhinitis include persistent or intermittent **nasal congestion** and **rhinorrhea** (runny nose). - These symptoms result from the dysregulation of the autonomic control over nasal vasculature and glands. *It involves autonomic dysfunction of nasal blood vessels* - This statement is **true** and describes the core mechanism of vasomotor rhinitis, where the **parasympathetic nervous system** is relatively overactive, leading to vasodilation and increased glandular secretions. - This dysfunction causes the characteristic symptoms without an allergic or infectious trigger. *It is triggered by non-allergic stimuli like weather changes and strong odors* - This statement is **true** as patients with vasomotor rhinitis often report symptoms triggered by **environmental irritants** such as strong perfumes, temperature changes, humidity fluctuations, or even emotional stress. - These triggers differentiate it clinically from allergic rhinitis.

Q: Saddle nose deformity is seen in?

Tertiary Syphilis. ***Tertiary Syphilis*** - **Saddle nose deformity** is a characteristic late manifestation of **tertiary syphilis** due to destructive lesions (gummas) affecting the nasal cartilage and bone [1], [2]. - It results from the **collapse of the nasal bridge**, leading to a flattened appearance [2]. *Primary Syphilis* - Characterized by a **chancre**, a painless ulcer, usually at the site of infection [1]. - This stage does not involve destructive lesions of the nose. *Secondary Syphilis* - Presents with a **widespread rash**, lymphadenopathy, and mucous patches [1]. - While systemic, it typically does not cause structural damage like saddle nose deformity. *Lupus Vulgaris* - This is a **cutaneous form of tuberculosis**, characterized by chronic, destructive skin lesions. - While it can cause facial disfigurement, saddle nose deformity is not a typical feature of lupus vulgaris.

Question 1

Which anatomical structure lies between the bulla ethmoidalis and the uncinate process of the ethmoid?

Accepted Answer

Ethmoidal infundibulum

Answer

Bulla ethmoidalis

Answer

Uncinate process of ethmoid

Answer

Maxillary sinus

Question 2

What lies between the middle and inferior turbinate?

Accepted Answer

Middle meatus

Answer

Superior meatus

Answer

Inferior meatus

Answer

Hiatus semilunaris

Question 3

What is the narrowest part of the nasal cavity?

Accepted Answer

Internal nasal valve area

Answer

Vestibule

Answer

Choanae

Answer

Inferior turbinate

Question 4

Which perforation of the tympanic membrane is most commonly seen with tubotympanic CSOM?

Accepted Answer

Central

Answer

Anterosuperior

Answer

Posterosuperior

Answer

Posteroinferior

Question 5

Most common cause of otitis externa is

Accepted Answer

Bacterial infection

Answer

Fungal infection

Answer

Seborrheic disease

Answer

Herpes Zoster

Question 6

Which of the following is a cause of objective tinnitus?

Accepted Answer

Carotid artery aneurysm

Answer

Impacted Wax

Answer

Meniere's disease

Answer

Ototoxic drugs

Question 7

Hitselberger's sign is seen in?

Accepted Answer

Acoustic neuroma

Answer

Glomus Tumour

Answer

Nasal angiofibroma

Answer

Acute suppurative otitis media

Question 8

Which of the following statements about vasomotor rhinitis is NOT true?

Accepted Answer

It is caused by an allergic reaction

Answer

It is due to parasympathetic overactivity

Answer

Resistant cases may need cryotherapy

Answer

It may lead to hypertrophic rhinitis

Question 9

Which of the following statements about vasomotor rhinitis is false?

Accepted Answer

It is an infective condition

Answer

It primarily presents with nasal congestion and rhinorrhea

Answer

It involves autonomic dysfunction of nasal blood vessels

Answer

It is triggered by non-allergic stimuli like weather changes and strong odors

Question 10

Saddle nose deformity is seen in?

Accepted Answer

Tertiary Syphilis

Answer

Primary Syphilis

Answer

Secondary Syphilis

Answer

Lupus Vulgaris

NEET-PG 2015

Anatomy

ENT

Internal Medicine