The arterial blood profile seen in compensated metabolic alkalosis is: pH: _____[HCO3-]: increasedPCO2: increased
Increased arterial pH (metabolic alkalosis) causes compensatory _____-ventilation with consequent increased PCO2
At _____ urine pH, the BH+ form of a weak base exists, thus increasing excretion
Un-reabsorbed _____ (15%) serves as a urinary buffer for H+
The increased _____ pH following gastric acid secretion is referred to as the 'alkaline tide' (e.g. after meals, vomiting)
_____ is also known as volatile acid
As pH of the tubular fluid _____, the excretion of H+ as NH4+ increases
The primary disturbance in respiratory acidosis is _____-ventilation, which results in increased CO2
In response to _____ pH sensed by chemoreceptors, the breathing rate increases (hyperventilation)
The primary disturbance in metabolic alkalosis is a(n) _____ in HCO3- OR a decrease in H+
Acid-Base Chemistry
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Respiratory Regulation of Acid-Base Balance
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Renal Regulation of Acid-Base Balance
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Bicarbonate Buffer System
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Non-Bicarbonate Buffer Systems
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Respiratory Acidosis and Alkalosis
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Metabolic Acidosis and Alkalosis
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Mixed Acid-Base Disorders
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Compensatory Mechanisms
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Clinical Assessment of Acid-Base Status
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