Laryngeal Disorders — MCQs
Reinke's layer is seen in:
A 45-year-old patient presents with persistent hoarseness for 3 months. Which finding on indirect laryngoscopy is most concerning for malignancy?
After a total thyroidectomy, the surgeon is unable to extubate the patient, who shows cyanosis and respiratory distress. What is the most likely cause of the inability to extubate?
A singer presents with difficulty singing at a high pitch. On examination, bowing of the vocal cord is observed on the right side. Which of the following muscles has likely been compromised?
During thyroidectomy, damage to which nerve leads to loss of high-pitched voice?
In an infant brought with stridor, diagnosed with laryngomalacia, which of the following is NOT typically observed?
Which of the following steps in thyroid surgery is least likely to result in hoarseness of voice?
Which of the following conditions is least likely to cause bilateral recurrent laryngeal nerve palsy?
All of the following are indications for tracheostomy except ?
Adenoidectomy is contraindicated in:
Laryngeal Disorders Indian Medical PG Practice Questions and MCQs
Question 1: Reinke's layer is seen in:
- A. Vocal cord (Correct Answer)
- B. Tympanic membrane
- C. Cochlea
- D. Reissner's membrane
Explanation: ***Vocal cord*** - **Reinke's space**, also known as Reinke's layer, is the superficial layer of the **lamina propria** of the vocal cord. - This layer is crucial for the **vibration of the vocal folds**, which is essential for sound production. *Tympanic membrane* - The **tympanic membrane**, or eardrum, is a thin membrane that separates the external ear from the middle ear [1]. [2]. - It vibrates in response to **sound waves** and transmits these vibrations to the ossicles, playing a role in hearing, not vocalization [1], [2]. *Cochlea* - The **cochlea** is a spiral-shaped cavity in the inner ear involved in the sense of hearing [2]. - It contains the **organ of Corti**, which converts sound vibrations into nerve impulses but has no connection to vocal cord anatomy [2]. *Reissner's membrane* - **Reissner's membrane** (vestibular membrane) is a thin membrane that separates the scala vestibuli from the scala media within the cochlea. - Its primary function is to maintain the **composition of the endolymph** in the scala media, and it is part of the auditory system, not the larynx.
Question 2: A 45-year-old patient presents with persistent hoarseness for 3 months. Which finding on indirect laryngoscopy is most concerning for malignancy?
- A. Reinke's edema
- B. Unilateral cord paralysis (Correct Answer)
- C. Bilateral polyps
- D. Vocal cord nodules
Explanation: ***Unilateral cord paralysis*** - **Unilateral cord paralysis** can be an indicator of an underlying malignancy impinging on the **recurrent laryngeal nerve**, which innervates the vocal cords. - The **persistent hoarseness** for 3 months, combined with paralysis, raises significant concern for a malignant process in the head, neck, or chest. *Reinke's edema* - **Reinke's edema** is typically associated with **chronic irritation** like smoking and presents as a swollen, gelatinous fluid collection in the superficial lamina propria. - While it causes hoarseness, it is a **benign condition** and not directly indicative of malignancy. *Bilateral polyps* - **Vocal cord polyps** are typically **benign lesions** often caused by vocal trauma or abuse, and while they can cause hoarseness, they are not usually a direct sign of malignancy, especially when bilateral. - While requiring management, polyps themselves do **not raise immediate concern for cancer** compared to paralysis. *Vocal cord nodules* - **Vocal cord nodules** (singer's nodules) are benign, bilateral lesions caused by **vocal abuse** and are a common cause of hoarseness. - They are a benign condition and do not suggest an underlying malignancy at their core.
Question 3: After a total thyroidectomy, the surgeon is unable to extubate the patient, who shows cyanosis and respiratory distress. What is the most likely cause of the inability to extubate?
- A. Bilateral recurrent laryngeal nerve palsy (Correct Answer)
- B. Unilateral recurrent laryngeal nerve palsy
- C. Superior laryngeal nerve palsy
- D. Hemorrhage
Explanation: ***Bilateral recurrent laryngeal nerve palsy*** - After total thyroidectomy, injury to both **recurrent laryngeal nerves** can lead to paralysis of the abductor muscles of the vocal cords causing them to approximate, leading to **airway obstruction**, cyanosis, and respiratory distress. - This condition prevents successful extubation and often necessitates **reintubation** or **tracheostomy**. *Unilateral recurrent laryngeal nerve palsy* - Causes **hoarseness** due to unilateral vocal cord paralysis but typically does not result in severe airway obstruction or inability to extubate. - The unaffected vocal cord can usually compensate sufficiently to maintain an adequate airway for breathing. *Superior laryngeal nerve palsy* - Primarily affects the **protective reflexes of the larynx** and vocal cord tension (pitch), leading to issues like **aspiration risk** and a weak, breathy voice. - It does not directly cause vocal cord paralysis in a position that obstructs the airway. *Hemorrhage* - While a significant **post-operative hemorrhage** in the neck can cause airway compression and respiratory distress, it usually manifests as **neck swelling** and possibly hypovolemic shock. - The scenario explicitly states "inability to extubate," suggesting a vocal cord issue rather than external compression by a hematoma.
Question 4: A singer presents with difficulty singing at a high pitch. On examination, bowing of the vocal cord is observed on the right side. Which of the following muscles has likely been compromised?
- A. Posterior cricoarytenoid
- B. Lateral cricoarytenoid
- C. Cricothyroid (Correct Answer)
- D. Thyroarytenoid
Explanation: ***Cricothyroid*** - The **cricothyroid muscle** is primarily responsible for **tensioning and elongating the vocal cords**, which is crucial for increasing vocal pitch. - Damage to this muscle or its innervation (superior laryngeal nerve) results in an inability to reach higher pitches and can cause **vocal cord bowing** due to reduced tension. *Posterior cricoarytenoid* - This muscle is the **primary abductor** of the vocal cords, meaning it opens the vocal cords for breathing. - Compromise would lead to difficulty breathing or a paralyzed vocal cord in the adducted position, not bowing with difficulty singing high notes. *Lateral cricoarytenoid* - The **lateral cricoarytenoid muscle** is a **vocal cord adductor** and rotator, bringing the vocal cords together to regulate voice intensity. - Dysfunction typically results in a weak and breathy voice, or difficulty bringing the cords together, not specifically difficulty with high pitch. *Thyroarytenoid* - The **thyroarytenoid muscle** (which includes the vocalis muscle) acts to **relax and shorten the vocal cords**, lowering pitch and modulating vocal cord tension. - Dysfunction would primarily lead to difficulty with lower pitches or a hoarse voice, as it prevents proper relaxation of the vocal cords.
Question 5: During thyroidectomy, damage to which nerve leads to loss of high-pitched voice?
- A. External branch of the superior laryngeal nerve (Correct Answer)
- B. Hypoglossal nerve
- C. Vagus nerve
- D. Recurrent laryngeal nerve
Explanation: ***External branch of the superior laryngeal nerve*** - The **external branch of the superior laryngeal nerve** innervates the **cricothyroid muscle**, which is responsible for tensing the vocal cords. - Damage to this nerve paralyzes the cricothyroid muscle, leading to an inability to tense the vocal cords, resulting in a **monotonous voice** and **loss of high-pitched tones**. *Hypoglossal nerve* - The **hypoglossal nerve (CN XII)** controls the muscles of the **tongue**, affecting articulation and swallowing, but not vocal pitch directly. - Damage primarily causes **tongue deviation** and **difficulty with speech (dysarthria)** and swallowing. *Vagus nerve* - The **vagus nerve (CN X)** gives rise to both the **superior laryngeal nerve** and the **recurrent laryngeal nerve**. - While damage to the vagus nerve trunk would affect vocalization, the question specifically asks about loss of high-pitched voice, which points to a more localized injury to one of its branches. *Recurrent laryngeal nerve* - The **recurrent laryngeal nerve** innervates most of the intrinsic laryngeal muscles, including the **thyroarytenoid** and **posterior cricoarytenoid muscles**, primarily affecting vocal cord adduction and abduction. - Damage typically causes **hoarseness** due to vocal cord paralysis, and in severe cases, difficulty breathing, but it does not specifically lead to the *loss of high-pitched voice* as directly as superior laryngeal nerve damage.
Question 6: In an infant brought with stridor, diagnosed with laryngomalacia, which of the following is NOT typically observed?
- A. Stridor will be inspiratory
- B. Hoarseness (Correct Answer)
- C. Prominent arytenoids
- D. Floppy aryepiglottic folds
Explanation: ***Correct: Hoarseness*** - **Laryngomalacia** primarily involves the collapse of supraglottic structures during inspiration, leading to inspiratory stridor - Hoarseness is NOT typically observed because laryngomalacia does **not directly affect the vocal cords** - Hoarseness indicates pathology at the level of the **vocal cords** themselves (such as vocal cord paralysis or inflammation), which is a different entity - The supraglottic collapse in laryngomalacia occurs above the vocal cords, leaving vocal cord function intact *Incorrect: Stridor will be inspiratory* - **Inspiratory stridor** is the hallmark feature of laryngomalacia - The collapse of supraglottic structures during inspiration creates a narrow airway, producing the characteristic high-pitched sound on inhalation - This is the most common presenting symptom in affected infants *Incorrect: Prominent arytenoids* - Laryngoscopy in laryngomalacia often reveals **prominent or redundant arytenoid mucosa** - The collapse of redundant tissue over the arytenoids makes them appear more prominent due to inward movement during inspiration - This contributes to the airway obstruction seen in the condition *Incorrect: Floppy aryepiglottic folds* - **Floppy, shortened aryepiglottic folds** are a hallmark anatomical feature of laryngomalacia - These folds collapse inward during inspiration, obstructing the laryngeal inlet - This collapse is the primary mechanism causing the inspiratory stridor in laryngomalacia
Question 7: Which of the following steps in thyroid surgery is least likely to result in hoarseness of voice?
- A. Ligation of superior thyroid artery
- B. Removal of the tubercle of Zuckerkandl
- C. Division of strap muscles (Correct Answer)
- D. Dissection of Beahrs triangle
Explanation: ***Division of strap muscles*** - Dividing the **strap muscles** (sternohyoid, sternothyroid, omohyoid) provides surgical access to the thyroid gland but does not directly involve structures critical for vocal cord function. - While it may cause temporary **neck discomfort** or altered neck contour, it is least likely to lead to hoarseness. *Ligation of superior thyroid artery* - Ligation of the **superior thyroid artery** occurs in close proximity to the **external laryngeal nerve**, a branch of the superior laryngeal nerve. - Damage to the external laryngeal nerve can cause subtle voice changes due to **cricothyroid muscle paralysis**, impacting pitch. *Removal of the tubercle of Zuckerkandl* - The **tubercle of Zuckerkandl** is a posterior extension of the thyroid gland, often lying close to the **recurrent laryngeal nerve**. - Its removal requires careful dissection in an area where the recurrent laryngeal nerve is vulnerable to **traction or direct injury**, which can cause hoarseness. *Dissection of Beahrs triangle* - **Beahrs triangle** is an anatomical landmark formed by the common carotid artery, inferior border of the thyroid lobe, and the recurrent laryngeal nerve. - Dissection within this triangle carries a high risk of **recurrent laryngeal nerve injury**, leading to vocal cord paralysis and significant hoarseness.
Question 8: Which of the following conditions is least likely to cause bilateral recurrent laryngeal nerve palsy?
- A. Thyroid carcinoma
- B. Lymphadenopathy
- C. Thyroid surgery
- D. Aortic aneurysm (Correct Answer)
Explanation: ***Aortic aneurysm*** - An aortic aneurysm, especially of the ascending aorta, is **less likely to cause bilateral recurrent laryngeal nerve palsy** because the left recurrent laryngeal nerve typically hooks under the aortic arch, while the right nerve hooks under the subclavian artery. - For **bilateral involvement**, two separate and simultaneous lesions affecting both nerves would be required at different anatomical locations with this etiology, making it a rare cause. *Thyroid carcinoma* - An aggressive **thyroid carcinoma** can directly invade or compress the recurrent laryngeal nerves (RLNs) due to their proximity to the thyroid gland. - If the carcinoma is extensive or multifocal, it can lead to **bilateral involvement** by affecting both nerves. *Lymphadenopathy* - Significant **cervical or mediastinal lymphadenopathy** (e.g., due to metastatic disease or lymphoma) can compress or encase both recurrent laryngeal nerves. - This proximity allows for potential **bilateral compression or damage** to the nerves as they ascend in the tracheoesophageal grooves. *Thyroid surgery* - **Thyroidectomy** is a common cause of recurrent laryngeal nerve injury due to the nerves' close anatomical relationship with the thyroid gland. - **Bilateral recurrent laryngeal nerve palsy** can occur if both nerves are damaged during dissection, often due to surgical misidentification, thermal injury, or traction.
Question 9: All of the following are indications for tracheostomy except ?
- A. Coma after head injury
- B. Maxillofacial injury
- C. Bilateral abductor palsy
- D. Superior laryngeal nerve palsy (Correct Answer)
Explanation: ***Superior laryngeal nerve palsy*** - While superior laryngeal nerve palsy can cause **hoarseness** and **dysphagia** due to impaired laryngeal sensation and cricothyroid muscle function, it typically does not directly lead to **airway obstruction** requiring a tracheostomy. - The primary concern with this condition is often **aspiration risk**, which is usually managed through compensatory swallowing techniques or dietary modifications, not surgical airway establishment. *Coma after head injury* - Patients in a **prolonged coma** or with severe **neurological impairment** often lose their protective airway reflexes (e.g., cough, gag reflex), increasing the risk of **aspiration** and making **pulmonary toilet** difficult. - A tracheostomy provides a secure, long-term airway for **ventilatory support**, suctioning, and protection against aspiration in these patients. *Maxillofacial injury* - Severe **maxillofacial trauma** can cause significant **airway obstruction** due to edema, hemorrhage, or anatomical disruption of the upper airway structures. - In such cases, a tracheostomy may be necessary to bypass the obstructed area and establish a **stable airway** for respiration. *Bilateral abductor palsy* - **Bilateral abductor palsy** results in failed abduction of both vocal cords, leading to a fixed, adducted position of the vocal cords that can cause severe or complete **airway obstruction**. - This condition is a direct and urgent indication for tracheostomy to ensure an **open airway**.
Question 10: Adenoidectomy is contraindicated in:
- A. SOM
- B. CSOM
- C. Bleeding disorder (Correct Answer)
- D. None of the options
Explanation: ***Bleeding disorder*** - Adenoidectomy involves surgical removal of tissue, which carries a risk of **intraoperative and postoperative bleeding**. - In individuals with a **pre-existing bleeding disorder**, this risk is significantly elevated, potentially leading to serious complications. *SOM* - **Serous otitis media (SOM)**, or otitis media with effusion, is often caused by Eustachian tube dysfunction, which can be exacerbated by adenoid hypertrophy. - Adenoidectomy can actually be a **treatment for recurrent SOM**, as it can relieve obstruction of the Eustachian tube. *CSOM* - **Chronic suppurative otitis media (CSOM)** involves a persistent perforation of the tympanic membrane with chronic ear discharge. - While adenoid hypertrophy can contribute to Eustachian tube dysfunction and recurrent acute otitis media that might lead to CSOM, an adenoidectomy is **not directly contraindicated** for CSOM itself. *None of the options* - This option is incorrect because **bleeding disorder** is a clear contraindication for adenoidectomy due to the increased risk of hemorrhagic complications.
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