Benign Paroxysmal Positional Vertigo

Benign Paroxysmal Positional Vertigo

Benign Paroxysmal Positional Vertigo

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BPPV Basics - Dizzy When I Do This!

  • Benign Paroxysmal Positional Vertigo (BPPV): Characterized by sudden, brief episodes of vertigo.
  • Etiology: Dislodged otoconia (calcium carbonate crystals, "canaliths") from utricle migrate into Semicircular Canals (SCCs).
    • Most common: Posterior SCC (PSC) involvement, termed canalithiasis.
    • Rarer: Cupulolithiasis (otoconia adhere to cupula).
  • Triggers: Specific head movements (e.g., rolling in bed, looking up).
  • Clinical: Vertigo lasts < 1 minute, often with nausea. Nystagmus present. No hearing loss or tinnitus. Inner ear anatomy and otoconia displacement in BPPV

⭐ BPPV is the most common cause of recurrent vertigo, especially in the elderly.

Pathophysiology - Loose Rocks Rolling

  • Otoconia (calcium carbonate crystals) from the utricular macula become dislodged.
  • These dense particles ("ear rocks") inappropriately enter the semicircular canals (SCCs).

    ⭐ The posterior semicircular canal is involved in 80-90% of BPPV cases due to its dependent position.

  • Mechanism of vertigo:
    • Canalithiasis (common, ~90%): Otoconia are free-floating in the SCC endolymph. Head movements cause them to move, creating endolymphatic flow that deflects the cupula, inducing vertigo and nystagmus.
    • Cupulolithiasis (rarer, ~10%): Otoconia adhere to the cupula, making it gravity-sensitive. This results in more persistent vertigo and nystagmus when the head is in provoking positions. Otoconia displacement in posterior canal

Clinical Picture & Diagnosis - Spin Doctor's Clues

  • Symptoms:
    • Sudden, brief vertigo (< 1 min), triggered by head movements (e.g., rolling in bed, looking up).
    • Nausea/vomiting common.
    • NO hearing loss, tinnitus, or focal neurological signs.
  • Diagnosis:
    • Dix-Hallpike Maneuver (Posterior Canal - most common):
      • Head turned 45°, then rapid supine movement with head extended 20-30°.
      • Positive: Latency, then torsional upbeating nystagmus (to affected ear) + vertigo; fatigable.
    • Supine Roll Test (Horizontal Canal):
      • Head rotation in supine position.
      • Nystagmus: Geotropic or apogeotropic.

⭐ The Dix-Hallpike maneuver elicits a characteristic nystagmus (e.g., torsional, upbeating, latency, fatigability) in posterior canal BPPV.

Dix-Hallpike Maneuver Steps

Management - Getting Stones Settled

  • Primary Goal: Reposition displaced otoconia from canals to utricle via Canalith Repositioning Procedures (CRPs). High success rates.
  • Key Maneuvers:
    • Epley Maneuver (Posterior Canal): (📌 "EPLEY = POSTERIOR PLEASES")

      ⭐ The Epley maneuver is highly effective (80-95% success rate) for treating posterior canal BPPV.

    • Semont Maneuver (Posterior Canal): Alternative for cupulolithiasis.
    • Barbecue Roll (Lempert) (Horizontal Canal): For geotropic type.
    • Gufoni Maneuver (Horizontal Canal): For apogeotropic type.
  • Post-CRP Care:
    • Avoid quick head movements (24-48 hrs).
    • Sleep head-elevated (45°, 1-2 nights).
  • Adjuncts:
    • Vestibular suppressants (e.g., dimenhydrinate) for severe nausea pre/post-CRP. Short-term. Epley Maneuver Steps Diagram Awaiting image generation for: Diagram illustrating Epley Maneuver steps

DDx & Prognosis - Not Always BPPV

  • DDx (Atypical? Consider):
    • Vestibular neuritis/labyrinthitis
    • Meniere's disease
    • Vertebrobasilar insufficiency (VBI)
    • Migrainous vertigo
    • Central causes (e.g., cerebellar)
  • Prognosis:
    • Excellent with Canalith Repositioning Maneuvers (CRMs).
    • High CRM success (e.g., Epley 80-95%).

    ⭐ While BPPV has a good prognosis with treatment, recurrence is common, affecting up to 50% of patients within 5 years.

    • Residual dizziness post-CRM possible.

High‑Yield Points - ⚡ Biggest Takeaways

  • BPPV is the most common cause of peripheral vertigo.
  • Caused by free-floating otoconia (canaliths) in semicircular canals, typically the posterior canal.
  • Characterized by sudden, brief episodes of vertigo (<1 minute) triggered by head position changes.
  • Dix-Hallpike maneuver is diagnostic, eliciting rotatory nystagmus with latency and fatigability.
  • Treatment involves Canalith Repositioning Maneuvers (CRMs) like the Epley maneuver.
  • Importantly, there is no associated hearing loss, tinnitus, or focal neurological deficits (distinguishing from other causes).

Practice Questions: Benign Paroxysmal Positional Vertigo

Test your understanding with these related questions

A 60-year-old man presents to his primary care physician complaining that he often feels as if the room is spinning when he gets up from a recumbent position or turns his head. He has not lost consciousness and has had no chest pain. He has no cardiac history, and a recent treadmill test showed no abnormalities. On examination, the sensation can be produced by rapidly turning the head. It can be reproduced many times, but it eventually ceases. Nystagmus is elicited. Hearing is normal. Which of the following is the MOST likely mechanism for this patient's symptoms?

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Flashcards: Benign Paroxysmal Positional Vertigo

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A normal person or one with sensorineural hearing loss hears _____ when the ear canal is occluded and softer when the canal is open (Bing positive)

TAP TO REVEAL ANSWER

A normal person or one with sensorineural hearing loss hears _____ when the ear canal is occluded and softer when the canal is open (Bing positive)

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