Adrenal disorders US Medical PG Flashcards - Medical Study Cards
Master Adrenal disorders with OnCourse flashcards. These spaced repetition flashcards are designed for medical students preparing for NEET PG, USMLE Step 1, USMLE Step 2, MBBS exams, and other medical licensing examinations.
Adrenal disorders Flashcard Deck - 10 Cards
Flashcard 1: What is the state of the adrenal glands in Cushing syndrome due to ACTH secreting pituitary adenoma (hyperplasia, atrophy; one, both)?_____
Answer: Hyperplasia of both adrenal glands
Flashcard 2: Conn syndrome presents with hypertension, hypokalemia, and metabolic alkalosis due to _____ aldosterone levels
Answer: increased
Flashcard 3: Does secondary adrenal insufficiency present with skin/mucosal hyperpigmentation?_____
Answer: No, because ACTH levels are decreased
Flashcard 4: Chronic glucocorticoid use may cause Iatrogenic _____ syndrome
Answer: Cushing
Flashcard 5: What is the most common cause of Cushing syndrome?
Answer: Exogenous glucocorticoids
Flashcard 6: _____ hyperaldosteronism is characterized by high aldosterone and high renin
Answer: Secondary
Extra:
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characterized by a aldosterone/renin ratio ~ 10 (renin causing high aldosterone)
Flashcard 7: One common cause of acute primary adrenal insufficiency is _____ syndrome
Answer: Waterhouse-Friderichsen
Flashcard 8: Kallmann syndrome
Answer: defective migration of GnRH cells
Extra: anosmia, lack of secondary sex characteristicsdecreased: GnRH, FSH, LH, testosterone, sperm count
Flashcard 9: Hormone profile in Hypogonadotropic Hypogonadism (LH/FSH and Sex Steroids)
Answer: Decreased LH/FSH and decreased Testosterone/Estrogen
Extra: Hypogonadotropic hypogonadism (secondary hypogonadism) is characterized by low gonadotropins (LH, FSH) which leads to low sex steroid production.
Contrast with Hypergonadotropic hypogonadism (primary): High LH/FSH, low sex steroids.
Flashcard 10: Hormonal profile in primary hypogonadism:
Answer: Increased LH, Decreased Testosterone
Extra: In primary hypogonadism (hypergonadotropic hypogonadism), the problem is at the level of the testes. Loss of negative feedback from testosterone leads to a compensatory rise in LH (and FSH).
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