Burns Management

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Burn Assessment - Sizing Up The Sizzle

  • TBSA Estimation:
    • Rule of Nines (Adults): Head/Neck 9%, Arm 9% (each), Trunk 18% (ant/post each), Leg 18% (each), Perineum 1%.
    • Lund-Browder Chart: Most accurate for children.

      ⭐ Lund-Browder chart is the most accurate method for estimating TBSA in children.

    • Palmer Method: Patient's palm ≈ 1% TBSA (scattered burns). Rule of Nines vs Lund-Browder Burn Charts
  • Burn Depth:
    • Superficial (1st°): Epidermis. Red, painful, no blisters. Heals 3-6d.
    • Partial-thickness (2nd°):
      • Superficial: Papillary dermis. Blisters, painful, blanches. Heals 7-21d.
      • Deep: Reticular dermis. Mottled, ↓pain, ±blanching. Scarring. Heals >21d.
    • Full-thickness (3rd°): All layers. Leathery, insensate, no blanching. Grafting.
    • 4th°: To muscle/bone. Burn Depth Diagram
  • Burn Severity Classification (ABA Criteria):
  • Key Factors for Severity: Inhalation (soot, hoarseness, facial burns), circumferential, elec/chem, trauma, age (<5/>50 yrs).

Pathophysiology & Inhalation - Body's Inferno

  • Systemic Effects: Burn → Systemic Inflammatory Response Syndrome (SIRS) (cytokines ↑).
    • Cardiovascular: ↓CO, ↑capillary permeability → Burn shock.
    • Metabolic: Hypermetabolism & catabolism.
    • Immune: Immunosuppression → ↑infection risk.
  • Jackson's Zones of Injury: Coagulation (central, irreversible necrosis), Stasis (surrounding, potentially salvageable), Hyperemia (outer, vasodilation, recovers).
  • Inhalation Injury: Major cause of mortality. Suspect with: facial burns, singed nasal hairs, soot in sputum/oropharynx, hoarseness, wheezing, history of enclosed space fire.
    • Upper airway: Supraglottic edema → airway obstruction.
    • Lower airway: Chemical tracheobronchitis, mucosal sloughing, ARDS.
    • Systemic toxicity: Carbon Monoxide (CO), Cyanide (CN).
    • 📌 Mnemonic (Signs of Inhalation Injury - BURNS): Blisters/burns to face/neck, Using accessory muscles, Rales/rhonchi/wheezing, Nasal hair singeing/soot, Stridor/hoarseness.
  • Diagnosis & Management:
    • Bronchoscopy: Gold standard for diagnosing lower airway injury.
    • Early intubation if airway compromise suspected (e.g., stridor, respiratory distress, GCS <8).
    • Administer 100% humidified O2.

Jackson's Burn Wound Model

⭐ Cherry-red skin colour in CO poisoning is a late and unreliable sign; always suspect CO poisoning with high carboxyhemoglobin (COHb) levels in patients from enclosed space fires, even with normal pulse oximetry.

Acute Management - Dousing The Flames

  • Primary Survey (ABCDE) 📌:
    • Airway: Secure early if inhalation injury suspected (e.g., facial burns, soot, hoarseness, stridor). Consider cricothyroidotomy if intubation fails.
    • Breathing: 100% humidified O₂. Monitor for respiratory distress; chest escharotomy if needed.
    • Circulation: Stop burning (cool water, not ice). IV access (2 large-bore cannulas, unburnt skin if possible). Commence fluid resuscitation.
    • Disability: Assess GCS, pupils. Rule out associated trauma.
    • Exposure/Environment: Remove clothing, keep patient warm to prevent hypothermia.
  • Fluid Resuscitation (Crystalloids - Ringer's Lactate preferred):
    • Parkland Formula: $4 \text{ml} \times \text{Body Wt (kg)} \times % \text{TBSA}$.
    • Administration: 1st half in first 8 hours from time of burn, 2nd half in next 16 hours.
    • Target Urine Output: Adults 0.5-1 ml/kg/hr; Children 1-1.5 ml/kg/hr.
    • Children also need maintenance fluids (D5RL or D5NS).

Adult Burn Management in the ED

⭐ The first half of the calculated Parkland fluid volume is administered in the first 8 hours from the time of burn injury, not from the time of hospital admission.

  • Adjuncts:
    • Analgesia: IV opioids (Morphine).
    • Tetanus prophylaxis.
    • NG tube: For burns >20% TBSA or intubated patients (prevents aspiration, ileus).
    • Stress ulcer prophylaxis (PPIs/H2 blockers).

Wound Care & Complications - Healing The Hurt

  • Wound Management:
    • Cleaning: Gentle (e.g., chlorhexidine). Debride necrotic tissue.
    • Dressings: Non-adherent, absorbent.
    • Topical Agents:
      • Silver Sulfadiazine (SSD): Painless. Risk: leucopenia, poor eschar penetration.
      • Mafenide Acetate: Good eschar penetration. Risk: pain, metabolic acidosis.
      • Silver Nitrate (0.5%): Risk: electrolyte imbalance (↓Na, ↓Cl), stains.
  • Complications:
    • Infection/Sepsis: Leading cause of late death.
    • Contractures: Prevent with splinting, physiotherapy.
    • Hypertrophic scars, Keloids.
    • Marjolin's Ulcer: SCC in chronic burn wound.
  • Escharotomy Indications (Circumferential Burns):
    • Absent distal pulses; Impaired capillary refill.
    • Chest: Respiratory compromise (SpO2 <90% on 100% O2).

⭐ Silver sulfadiazine is widely used but can cause transient leucopenia; Mafenide acetate penetrates eschar well but can cause metabolic acidosis due to carbonic anhydrase inhibition.

High‑Yield Points - ⚡ Biggest Takeaways

  • Parkland formula (4ml x %TBSA x Wt) guides fluid resuscitation: 50% in first 8 hrs, remainder in next 16 hrs.
  • Rule of Nines for adult TBSA estimation; Lund-Browder chart is more accurate, especially for children.
  • Escharotomy is indicated for circumferential full-thickness burns causing compartment syndrome or respiratory compromise.
  • Suspect inhalation injury with facial burns, singed nasal hairs, or sooty sputum; early intubation is critical.
  • Silver sulfadiazine (SSD) is a common topical antimicrobial; Mafenide acetate penetrates eschar but is painful.
  • Electrical burns: high risk of arrhythmias (monitor ECG) and rhabdomyolysis (myoglobinuria).
  • Chemical burns: immediate copious water irrigation is key; identify the specific agent if possible (exceptions exist).

Practice Questions: Burns Management

Test your understanding with these related questions

Fluid given in first 8 hours to a 28 years old woman with 50 kg weight having burns on both lower limbs?

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Flashcards: Burns Management

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The primary cause of early mortality in burns is _____

TAP TO REVEAL ANSWER

The primary cause of early mortality in burns is _____

burn shock.

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