Fracture Complications

Fracture Complications

Fracture Complications

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Systemic Crises - Code Red Alerts

  • Hypovolemic Shock:

    • Common in polytrauma. ATLS classification (Class I-IV based on blood loss %: <15%, 15-30%, 30-40%, >40%).
    • Management: ABCDE, O₂, IV fluids (crystalloids, blood products).
  • Fat Embolism Syndrome (FES):

    • Long bone/pelvic fractures. Pathophysiology: Mechanical obstruction & chemical pneumonitis.
    • Gurd's Major Criteria (≥2): Respiratory distress, cerebral signs, petechial rash.
    • ⭐ > Classic triad of Fat Embolism Syndrome: respiratory distress, neurological symptoms, and petechial rash.
    • Prevention: Early fracture fixation. Management: Supportive (O₂, ventilation).
  • Venous Thromboembolism (VTE - DVT/PE):

    • Virchow's Triad: Stasis, hypercoagulability, endothelial injury.
    • Risk factors: Immobility, surgery, prior VTE. Wells Score (e.g., DVT >3 high risk).
    • Prophylaxis: Mechanical (SCDs), Pharmacological (LMWH, e.g., Enoxaparin 40mg OD).
    • Diagnosis: Doppler US (DVT), CTPA (PE). Treatment: Anticoagulation (Heparin, Warfarin).

Local Limb Threats - Pressure Cookers

  • Compartment Syndrome: ↑ Pressure in fascial compartment → ↓ tissue perfusion, ischemia.
    • 📌 6 Ps: Pain (key, on passive stretch), Paresthesia, Pallor, Pulselessness (late), Paralysis.
    • Dx: Clinical; Intracompartmental pressure (ICP) > 30-45 mmHg.

    ⭐ A delta pressure (Diastolic BP - Compartment Pressure) of < 30 mmHg is a strong indication for fasciotomy in compartment syndrome.

    • Tx: Urgent Fasciotomy. Causes of Compartment Syndrome Venn Diagram
  • Acute Vascular Injury:
    • Hard signs (pulsatile bleed, absent pulse, thrill); Soft signs (↓ pulse, history).
    • Dx: ABI < 0.9; Doppler; CT Angio. Tx: Surgical repair.
  • Nerve Injury:
    • Seddon: Neuropraxia, Axonotmesis, Neurotmesis.
    • Sunderland: Grades I-V.
  • Crush Syndrome: Muscle crush → rhabdomyolysis.
    • Features: Myoglobinuria (dark urine), hyper$K^+$, ↑CK, AKI.
    • Tx: Aggressive IV fluids, urine alkalinization.

Healing Hijacked - Union & Infection

  • Fracture Infection (Osteomyelitis):
    • Timing: Acute (<2 wks), Chronic (>2 wks).
    • Signs: Local pain, swelling, erythema, discharge; systemic fever.
    • Diagnosis: ↑ESR/CRP, WBC; X-ray (sequestrum, involucrum), MRI; biopsy/culture.
    • Classifications: Cierny-Mader (anatomic type), Gustilo-Anderson (open #).
    • Management: Surgical debridement, targeted antibiotics, stable fixation.
  • Delayed Union:
    • Fracture not healed in expected timeframe (e.g., 3-6 months).
    • Causes: Infection, ↓blood supply, instability, poor nutrition, systemic factors.
  • Non-union:
    • No healing signs for 6-9 months or no progress for 3 consecutive months.
    • Types (Weber-Cech Classification):
      • Hypertrophic ('elephant foot'): Good biology, poor stability.
      • Atrophic ('pencil point'): Poor biology, ↓vascularity.
      • Oligotrophic: Viable, minimal/no callus, often due to large gap.

    ⭐ Hypertrophic non-union typically shows abundant 'elephant foot' callus, indicating good biology but poor stability, whereas atrophic non-union shows no callus, indicating poor biology.

    • Management: ORIF, bone grafts (autograft/allograft), Ilizarov, BMPs, ESWT.
  • Malunion:
    • Fracture healed in non-anatomical/unacceptable position.
    • Causes: Inadequate reduction or unstable fixation.
    • Leads to: Functional impairment, deformity. Management: Corrective osteotomy.

Forearm fracture non-union with plate fixation

Chronic Aftermath - Lasting Limb Woes

  • Avascular Necrosis (AVN)
    • ↓Blood supply → bone death. Common sites: Femoral head, scaphoid, talus.

      ⭐ The most common sites for avascular necrosis following a fracture are the femoral head (after neck of femur fracture), scaphoid waist, and talar neck.

    • Classifications: Ficat-Arlet (X-ray), Steinberg (MRI). Imaging: X-ray (crescent sign), MRI (best early).
    • Management: Core decompression, osteotomy, arthroplasty.
  • Post-Traumatic Osteoarthritis (PTOA)
    • Causes: Articular cartilage damage, joint incongruity. Prevention: Anatomic fracture reduction.
    • Management: NSAIDs, physiotherapy, arthroplasty.
  • Complex Regional Pain Syndrome (CRPS)
    • Types: Type I (no direct nerve injury), Type II (known nerve injury). Diagnosis: Budapest criteria.
    • Stages: 1 (Acute), 2 (Dystrophic), 3 (Atrophic).
    • Management: Multidisciplinary (physiotherapy, medications, sympathetic blocks).
  • Myositis Ossificans
    • Pathophysiology: Heterotopic ossification in muscle. Common sites: Elbow, thigh. Brooker classification.
    • Prevention: Gentle ROM, NSAIDs. Management: Observation; excise if mature (typically >6-12 months) & symptomatic.
  • Joint Stiffness & Contractures
    • Causes: Intra-articular adhesions, capsular fibrosis. Prevention: Early mobilization, physiotherapy.

AVN of femoral head: MRI, CT, and X-ray

High-Yield Points - ⚡ Biggest Takeaways

  • Compartment syndrome is a limb-threatening emergency; immediate fasciotomy is crucial.
  • Fat Embolism Syndrome classic triad: petechial rash, hypoxemia/respiratory distress, and neurological dysfunction.
  • Avascular Necrosis (AVN) commonly affects femoral head/neck, scaphoid (proximal pole), and talus body.
  • Nonunion risk factors include infection (osteomyelitis), poor vascularity, inadequate stabilization, and smoking.
  • Malunion is fracture healing in an anatomically incorrect position, leading to deformity.
  • Delayed union: fracture healing slower than expected for that specific bone.

Practice Questions: Fracture Complications

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Flashcards: Fracture Complications

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