Introduction to TE - Sudden Shed Show
Telogen Effluvium (TE) is diffuse, non-scarring hair shedding. Stressors prematurely shift hairs from the growth to the resting phase.
- Normal Hair Cycle Phases: 📌 All Cats Talk
- Anagen (Growth): 85-90% of hairs; lasts 2-6 years.
- Catagen (Transition): <1% of hairs; lasts 2-3 weeks.
- Telogen (Resting): 10-15% of hairs; lasts 2-3 months, then sheds.
- TE Disruption: ↑ Hairs prematurely enter telogen. Shedding typically follows 2-4 months after a trigger.

⭐ Normally, 10-15% of scalp hairs are in the telogen phase. In TE, this can increase to >30%, sometimes up to 50%.
TE Etiology - Trigger Happy Hair
Onset typically 2-4 months post-trigger.
Common Triggers:
- Physiological stress: Surgery, high fever (e.g., COVID-19, typhoid), severe illness.
- Psychological stress: Major life events.
- Postpartum period.
- Nutritional deficiencies: ↓Iron, ↓zinc, ↓biotin, ↓protein.
- Endocrinopathies: Thyroid disorders (hypo- or hyperthyroidism).
- Medications: Beta-blockers, anticoagulants, retinoids, lithium, OCP discontinuation.
- Crash diets / Rapid weight loss.
📌 TELOGEN Mnemonic for Triggers:
- Thyroid disorders
- Emotional/Psychological stress
- Low iron/nutrition (Fe, Zn, Biotin, Protein)
- Obstetrical (Postpartum)
- Germs (Severe illness/fever)
- Endocrine (other than thyroid)
- New medications / Nutritional crash
⭐ Postpartum hair loss is a classic, self-limiting form of telogen effluvium, usually resolving within 6-9 months postpartum without specific treatment beyond reassurance and nutritional support if needed (e.g., iron supplementation for deficiency).
TE Clinical & Diagnosis - Spotting the Shed
- Presentation: Acute, diffuse, non-scarring hair shedding. Onset typically 2-4 months post-trigger.
- History: Key to identify trigger (e.g., illness, stress, medication, nutritional deficiency).
- Hair Pull Test:
- Gently tug ~40-60 hairs.
- Positive: >10% of hairs (or >4-6 hairs) easily extracted. Hairs show club-shaped roots.
- Scalp Exam: Usually normal; no significant inflammation, scaling, or scarring.
- Trichoscopy Findings:
- Increased telogen (club) hairs.
- Empty follicular openings.
- Upright regrowing hairs (short, tapered).
⭐ Telogen hairs are characterized by a club-shaped root without a gelatinous sheath.
TE Differentials & Management - Ruling Out & Reassurance
- Key Differentials:
- Vs. AGA & AA:
Feature TE AGA AA Shedding Acute, diffuse Gradual, patterned Sudden, patchy Signs +ve Pull test Miniaturization Exclamation hairs - Others: Chronic TE (CTE), Anagen Effluvium.
- Vs. AGA & AA:
- Management:
- Reassure (self-limiting). Identify & correct triggers.
- Nutritional support (if deficient).
- Topical minoxidil (may shorten telogen; controversial for acute TE).
- Prognosis: Good; recovery 6-12 months post-trigger removal.
⭐ Unlike androgenetic alopecia, telogen effluvium does not typically cause miniaturization of hair follicles or a receding hairline.
High‑Yield Points - ⚡ Biggest Takeaways
- Diffuse, non-scarring hair shedding occurring 2-4 months after a stressful trigger.
- Triggers include childbirth, high fever, surgery, severe emotional stress, crash diets.
- Mechanism: Premature shift of anagen hairs to telogen phase.
- Positive hair pull test (gentle pull yields >4-6 hairs).
- Biopsy shows ↑ telogen hairs (>20-25%); no significant inflammation.
- Typically self-resolving within 6-12 months once trigger is addressed.
- If shedding persists >6 months, consider Chronic Telogen Effluvium.
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